tag:blogger.com,1999:blog-3364570834099131201.post9190909979277446080..comments2024-03-23T08:57:50.965-04:00Comments on ECG Interpretation: ECG Blog #59 — Giant T - Ischemia -Yamaguchi ECG Interpretationhttp://www.blogger.com/profile/02309020028961384995noreply@blogger.comBlogger2125tag:blogger.com,1999:blog-3364570834099131201.post-16467145879614282392018-03-30T20:11:46.714-04:002018-03-30T20:11:46.714-04:00@ MG — My impressions are: i) that the ECG may be ...@ MG — My impressions are: i) that the ECG may be highly variable for HCM — ranging from LBBB, LVH, RBBB, pseudo-MI patterns, prominent septal forces (tall anterior R waves and/or deep lateral or inferior Q waves) — to being NORMAL; and ii) that Apical CM is not any more “predictive” from ECG than is HCM — therefore distinction between the 2 is not something that can readily be done by ECG. As to your 2nd question — the ST elevation is isolated to lead III, and the overall pattern seen on this ECG is dominated by diffuse and very deep T wave inversion — so to me, the ST elevation does not weigh into my decision for justifying calling the cath lab. As per the theme of this Blog #59 — there is a big differential diagnosis — so history and exam and close follow-up are the factors that would determine if the cath lab should or should not be activated.ECG Interpretationhttps://www.blogger.com/profile/02309020028961384995noreply@blogger.comtag:blogger.com,1999:blog-3364570834099131201.post-69376294302569984952018-03-30T11:19:47.423-04:002018-03-30T11:19:47.423-04:00Hello Professor.
1)Can you tell us more about diff...Hello Professor.<br />1)Can you tell us more about differentiating between <br />Apical CM and HCM?<br />2)If this patient presented with chest pain without prior ECGs for comparison and in view of ST elevations in Lead 3 and ST depressions and Twave inversions in aVL, will activating cath lab justified??MGhttps://www.blogger.com/profile/06233522417024317416noreply@blogger.com