- How would YOU interpret the rhythm?
- Why are there pauses in rhythm strip "B"?
Figure-1: These 2 rhythm strips were both obtained from a 50-year old woman with palpitations. Not much time passed between the recording of rhythm strip "A" and rhythm strip "B". |
- That said — We are not told if this patient received any treatment. This is relevant — because there is an obvious difference between the 2 tracings.
As always — I began my approach to rhythm assessment with review of the Ps, Qs and 3Rs (as discussed in detail in ECG Blog #185).
- To emphasize — When applying the Ps, Qs, 3R System — You do not have to go in sequence. Instead — I favor review of whichever parameters are easiest to assess.
- The rhythm in "A" is fast and regular. The rate is ~135/minute. Although we only see a single monitoring lead — the QRS appears to be narrow. Sinus P waves do not appear to be present (at least not with a normal PR interval) — there is no obvious "relationship" between atrial activity and the QRS complex.
- IMPRESSION: The rhythm in "A" is a Regular SVT (SupraVentricular Tachycardia) at a rate of ~135/minute, without clear sign of sinus P waves.
PEARL #1: Recognition that the rhythm in Figure-1 is a Regular SVT without clear sign of sinus P waves (ie, without a definite upright P wave in lead II) — should prompt consideration of the following differential diagnosis LIST:
- i) Sinus Tachycardia (IF there is a possibility that sinus P waves might be hiding within the preceding ST-T wave);
- ii) A Reentry SVT (either AVNRT if the reentry circuit is contained within the AV node — or AVRT if an AP [Accessory Pathway] located outside the AV node is involved);
- iii) Atrial Tachycardia (ATach);
- iv) Atrial Flutter (AFlutter) with 2:1 AV conduction.
KEY Point: Although other entities may also produce a regular SVT (ie, sinoatrial node reentry tachycardia, junctional tachycardia) — they are far less common in practice. Therefore, remembering to think of the 4 entities in the above LIST whenever you encounter a regular SVT rhythm without clear sign of sinus P waves — will greatly facilitate determining the correct diagnosis.
- In Rhythm "A" — any of the above 4 entities could be present. The clinical reality — is that treatment of this rhythm will need to begin before we know for certain which of these entities is the actual diagnosis. That said — initial management of this patient will be similar regardless of which of the above 4 entities the rhythm happens to be (ie, consideration of a vagal maneuver — and/or use of an AV nodal blocking agent such as Adenosine, Verapamil-Diltiazem, or a ß-Blocker).
- There are pauses in Rhythm B.
- There is group beating (ie, groups of 2 beats — and of 3 beats — each separated by pauses that for the most part are of similar duration).
- P waves are now clearly present in Rhythm B.
- Beyond the Core: An important advanced observation that can be made with the use of calipers — is that the rate of the P waves that is seen in B (taking the P-P distance between any 2 P waves that are seen in a row) — is slightly slower than the ventricular rate in Rhythm A.
- This finding is relevant — because we can suspect that perhaps this patient was started on some type of AV nodal blocking agent in A — which resulted in slight slowing of the atrial rate, and development of some form of AV block ... But let's work up to this in step-by-step fashion ...
- PEARL #3: The reason we know there is conduction of at least some of the P waves that we see in Rhythm B — is that the identical PR interval is seen before most of the beats that end a pause! (RED arrows in Figure-2). This is not by chance! Instead — it tells us that the first beat in each group is being conducted, albeit with a long PR interval (ie, ~0.44 second).
Figure-2: The first beat in each group is being conducted, albeit with a long PR interval (RED arrows). |
- NOTE: PINK arrows highlight the 2 P waves in this rhythm strip that we don't see — because these P waves are hidden within the QRS of beats #2 and 7. But given how regular all other RED arrow P waves are — we know that on-time P waves are almost certain to be hiding under those PINK arrows!
- This suggests that the underlying rhythm in Figure-3 — is ATach (Atrial Tachycardia) at ~130/minute (RED and PINK arrows showing regular P waves throughout the rhythm strip).
- Recognition of group beating in Figure-2 — in which there is an underlying regular atrial rhythm + identical PR intervals that repeat + similar R-R interval duration for most of the pauses — strongly suggests that there is AV Wenckebach conduction.
- RED arrows highlight the initial P wave in most of the groups. Note that the PR interval of these P waves (ie, the PR interval that precedes beats #4, 6, 9, 11, and 14) — is identical (ie, prolonged to ~0.44 second in duration).
- Dark BLUE arrows highlight the 2nd P wave in 6 of the groups. The PR interval of these P waves (that precede beats #2, 5, 7, 10 and 12) — is longer than the PR interval highlighted by the RED arrows (ie, ~0.50 vs 0.44 second).
- Light BLUE arrows highlight the location of where a 3rd P wave is hidden in 2 of the groups. The PR interval of these P waves is longer still (ie, ~0.54 second).
- YELLOW arrows highlight the last P wave in a given group. These YELLOW P waves are not conducted because of the Wenckebach block. Note that after each of these YELLOW arrows — a RED arrow follows, thereby beginning the next group.
Figure-4: To facilitate appreciation of Wenckebach conduction — I've color-coded the P waves in this rhythm (See text). |
- I've kept the color-coding shown in Figure-4.
- It's easiest to follow events in the laddergram — by picking any group of beats, and starting by following the path of the RED arrow P wave in that group. For example — the RED arrow P wave before beat #6 manifests progressive PR interval lengthening for the dark BLUE and then light BLUE arrows that follow — until the YELLOW arrow P wave (that appears just before beat #8) is not conducted.
- The next Wenckebach group then begins with the RED arrow P wave that precedes beat #9.
- Wenckebach conduction in Figure-5 manifests 4:3 and 3:2 AV conduction.
- NOTE: The PR interval of the PINK arrow P wave is slightly longer than the PR interval for all of the RED arrow P waves. I'm not quite sure why. I drew the laddergram to reflect that this PINK arrow P wave is being conducted to the ventricles (but I added a question mark to acknowledge that I am not certain this P wave is conducted). That said — this does not matter clinically, because the "theme" of this rhythm is unchanged (ie, ATach with Wenckebach conduction).
- Now that we've established that Rhythm B represents ATach with 4:3 and 3:2 AV Wenckebach conduction — it should be apparent that the Rhythm A was ATach with 1:1 AV conduction and a long and constant PR interval (of ~0.32 second). What was thought to be a "pointed" T wave in Rhythm A — turned out to be the P wave of an ectopic Atrial Tachycardia.
- Putting It All Together: We were not told if today's patient received any treatment in between the time that Rhythm A and Rhythm B were recorded. I strongly suspect that some form of AV nodal blocking treatment was administered (ie, Adenosine, Verapamil-Diltiazem or a ß-Blocker) because: i) The ventricular (and therefore the atrial) rate of Rhythm A is slightly faster than the atrial rate in Rhythm B (ie, ~135/minute vs 130/minute); ii) The PR interval for the 1st conducted beat in each of the Wenckebach groupings in Rhythm B is longer than the PR interval during 1:1 AV conduction in Rhythm A (ie, 0.44 second — compared to 0.32 second); and, iii) Wenckebach conduction is seen in Rhythm B, whereas there was 1:1 AV conduction in Rhythm A.
- The most logical conclusion is that some AV nodal blocking agent was administered to the patient when she presented in Rhythm A — and that this resulted in slight slowing of the atrial rate, an increase in the PR interval — and Wenckebach conduction in which every 3rd or 4th beat was blocked.
Figure-7: I've labeled selected P waves in today's original tracings . |
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Acknowledgment: My appreciation to Hafiz Abdul Mannan Shahid (from Lahore, Pakistan) for the case and these tracings.
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ADDENDUM (1/6/2023):
- The Audio Pearls below may be helpful in assessment of the rhythms in today's case.
ECG Media PEARL #51a (7:40 minutes Audio) — Reviews of "Some Simple Steps to Help Interpret Complex Rhythms" ).
ECG Media PEARL #55 (4:20 minutes Audio) — What does the term, "SVT" mean? This Audio Pearl reviews the semantics and clinical application involved in use of this term.
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Relevant ECG Posts to Today's Case:
- ECG Blog #185 — My Systematic Approach to Rhythm Interpretation.
- ECG Blog #261 — A case of ATach with Wenckebach conduction.
- ECG Blog #55 — Acute inferior MI + AV Wenckebach.
- ECG Blog #154 — Acute inferior MI + AV Wenckebach.
- ECG Blog #168 — Acute inferior MI + Wenckebach (dual-level) block.
- ECG Blog #224 — Acute inferior MI + AV Wenckebach.
- ECG Blog #240 — Reviews assessment of the Differential Diagnosis of the Regular SVT Rhythm.
- ECG Blog #261 — Reviews assessment of a similar case to Rhythm "B".
- ECG Blog #229 — Why is AFlutter so commonly overlooked?
- ECG Blog #137 — AFlutter with an unusual conduction ratio.
- ECG Blog #138 — AFlutter vs Atrial Tachycardia.
- ECG Blog #40 — Another regular SVT that turned out to be AFlutter.
- González-Torrecilla et al: Ann Noninvasive Electrocardiol 16(1):85-95, 2011 — Reviews distinction between AVNRT vs AVRT and other regular SVT rhythms in patients without WPW.
- ECG Blog #188 — How to Read (and Draw) Laddergrams.
thank you for your explanation! I had a same EKG case but I couldn't interpret it like you!
ReplyDeleteTHANK YOU for the kind words! Glad my explanation was helpful! — :)
DeleteWow. That was brilliant. Classic KG Genius. I don't think anyone else could have solve this riddle, thanks Prof
ReplyDeleteTHANKS for the kind words. KEY = Use of calipers — :)
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