How would YOU interpret the tracing in Figure-1 — that was sent to me without additional information?
QUESTIONS:
- What is the rhythm?
- Is there AV dissociation? If so — WHY?
- Clinically — HOW would you approach interpretation of this tracing in optimal time-efficient fashion?
KEY Clinical Point:
If I was the medical provider charged with the care of the patient whose ECG is shown in Figure-1 — I would approach this tracing in the following sequential stages:
- I’d first establish that the patient was hemodnamically stable with this ECG and this cardiac rhythm. Assuming this patient is at least momentarily stable — I’d then proceed as follows:
- I’d take a quick look at the long lead rhythm strip.
- To EMPHASIZE: The rhythm shown in the long lead II (below the 12-lead tracing in Figure-1) is not a simple arrhythmia — so rather than trying to come up with a precise interpretation of the rhythm — I’d spend no more than 5-to-10 seconds to ensure there is a reasonable, stable-looking heart rate (which there is in Figure-1 — as the rhythm is supraventricular [narrow QRS everywhere] — some P waves are seen — and, a pattern of repetitive group beating (groups of 3 beats), with an average heart rate in the 60-80/minute rate range — appears to be present).
- I’d then spend no more than 5-to-10 seconds looking at the rest of the 12-lead ECG to ensure that there is no ongoing emergency in need of immediate care (ie, No acute MI; No hyperkalemia, etc.).
COMMENT:
While I always advocate a systematic approach to both rhythm and 12-lead ECG interpretation — optimal clinical ECG interpretation entails prioritizing those parts of the tracing in front of you that mandate your immediate attention. Later, when you have a moment of time — You can complete your systematic assessment.
- With Regard to the Cardiac Rhythm: Our initial 5-to-10 second assessment of the long lead II rhythm strip should be all that is needed at this instant in time — because this limited amount of time should still allow you to establish: i) That the patient is hemodynamically stable; and, ii) That the rhythm is supraventricular — with at least some P waves, a narrow QRS everywhere — and, an overall heart rate in the 60-80/minute range.
- PEARL #1 (Advanced Point): The fact that the QRS complex is narrow, with at least some P waves — and a pattern suggesting group beating — should clue us into: i) The possibility of Wenckebach conduction! — and, ii) That we need to carefully inquire about recent chest pain, paying special attention to the rest of this 12-lead ECG (looking carefully for signs of acute or recent infarction — because AV Wenckebach is common with acute inferior MI). It is much easier to identify Wenckebach conduction IF you are looking for it!
Regarding the 12-Lead ECG:
Applying the systematic approach I favor for 12-lead ECG interpretation (as detailed in ECG Blog #205):
- Rate & Rhythm: As stated above — there is a regular, supraventricular rhythm with some P waves, group beating, and an acceptable overall ventricular rate between ~60-80/minute.
- Intervals (PR-QRS-QTc): The PR interval varies — the QRS is narrow — the QTc looks normal (Best seen in leads V2,V3).
- Axis: Normal (about +60 degrees).
- Chamber Enlargement: Depending on age of the patient — voltage for LVH may be present (ie, Deepest S in V1,V2 + tallest R in V5,V6 ≥35 mm — IF the patient is ≥35 years of age).
- Q waves: Small q waves of uncertain significance are seen in the inferior leads.
- R Wave Progression: Normal, with transition (where height of the R wave becomes taller than the S wave is deep) — occurs normally, here by lead V4.
- ST-T wave Changes: Show some nonspecific ST-T wave flattening, and perhaps shallow T wave inversion in lead aVL. The slight amount of ST elevation that we see in lead V2 is common and normal.
- Clinical IMPRESSION: The ECG in Figure-1 shows nonspecific ST-T wave abnormalities — but nothing that looks acute.
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Looking Closer at the Rhythm in Today's CASE:
As reviewed in ECG Blog #185 — I favor the Ps, Qs, 3R Approach for systematic, time-efficient interpretation of the cardiac rhythm.
- PEARL #2: When looking for P waves — I find it easiest to first identify those P waves that I am certain about. I have done this with RED arrows in Figure-2.
- The presence of multiple, regularly-spaced RED ( = definitely present) P waves in Figure-2 suggests: i) that there is a very good chance that the underlying atrial rhythm will be regular throughout the long lead II rhythm strip; and, ii) that the P-P interval between any 2 consecutive RED arrows will clue us in to where the remaining (partially hidden) P waves can be found.
- Use of calipers can facilitate confirming that the underlying atrial rhythm in Figure-2 is in fact regular. With calipers — it takes only seconds to confirm this!
- PINK arrows highlight that the on-time beginning little "hump" at the very onset of the QRS of beats #2 and #5 clearly represents 2 additional on-time P waves.
- Completion of our "P wave search" can then be easily accomplished by noting the subtle-but-unmistakeable distortion under the PURPLE arrows toward the end of the T waves of beats #1 and 4 — and at the very onset of the QRS of beats #8 and #11.
Figure-2: I've labeled atrial activity in today's tracing with colored arrows (See text). |
PEARL #3: Recognition that true group beating is present, in association with an underlying regular atrial rhythm — strongly suggests there is some type of 2nd-degree AV block in which there is Wenckebach conduction (ie, the presence of an underlying regular P wave rhythm is an essential component of the typical forms of AV block).
- Focus on this concept facilitates recognizing the 3 true groupings in Figure-3 (within the dotted WHITE rectangles). Note the uncanny similarity in duration of the R-R interval between the first 2 beats in each group (ie, the R-R interval between beats #2-3; 5-6; and 8-9) — as well as for the R-R interval between the last 2 beats in each group (ie, the R-R interval between beats #3-4; 6-7; and #9-10) — as well as for duration of the R-R intervals of the short pauses that separate each of the groups (ie, the R-R interval between beats #1-2; 4-5; 7-8; and 10-11). This amount of symmetry for the relative duration of R-R intervals within each group of beats is not the result of chance!
Figure-3: RED arrows highlight that the atrial rhythm in the long lead II rhythm strip is regular. Note the 3 true groupings (within the dotted WHITE rectangles).
- Since beats #2,5,8 and 11 all manifest a narrow QRS complex — and are not preceded by sinus P waves with a realistic chance of conducting — this means that these 4 QRS complexes are junctional beats.
- Note that each of these beats is preceded by a similar R-R interval of just under 5 large boxes — which corresponds to an ever-so-slightly accelerated junctional escape rate of ~65/minute.
- That said — We have no idea if the WHITE-arrow P waves that appear at the onset of the QRS of beats #2,5,8 and 11 might possibly have conducted — IF they were given a chance to do so (ie, IF the junctional escape rate would have been slower than 65/minute).
- Most of the time when there is complete AV block — the escape rate (be this a junctional or ventricular escape rhythm) will be regular. The obvious irregularity of the ventricular response in Figure-4 — immediately tells us this is not complete AV block.
- QRS complexes that occur earlier-than-expected are likely to be conducted beats! (ie, Beats #4,7 and 10 in Figure-4 are therefore likely to be conducted — and beats #3,6,9 and 12 may also be conducted).
PEARL #6: As per PEARL #4 — we have already established that there is transient AV dissociation in Figure-4 (because beats #2,5,8,11 are clearly not conducted). There appear to be 2 reasons for this transient AV dissociation in today's rhythm:
- As reviewed in ECG Blog #192 — the 3 Causes of AV Dissociation are: i) Default; ii) Usurpation; and, iii) Some form of 2nd- or 3rd-degree AV block.
- Today's tracing is interesting — in that the reason for transient AV dissociation (ie, non-conduction of the WHITE-arrow P waves in Figure-4) — appears to be a combination of the slightly accelerated junctional beats (beats #2,5,8 and 11) — and the 2nd-degree AV block that leads to the slight pauses after beats #1,4,7 and 10.
- As already stated — the WHITE-arrow P waves in each group do not conduct to the ventricles — because the PR interval is too short.
- That said — We know that the next P wave in each grouping (ie, the light BLUE-arrow P waves) — does conduct to the ventricles, because the PR interval preceding beats #3,6,9 and 12 are all equal!
- This is followed by the dark BLUE-arrow P waves that are also conducting — but with slight increase in the PR interval compared to the light BLUE PR interval. Once again — We know that the dark BLUE-arrow P waves are conducting — because the PR interval preceding beats #4,7 and 10 are all equal!
- The last P wave in each grouping (ie, the YELLOW-arrow P waves) — are non-conducted — thus completing the AV Wenckebach cycle of progressive PR interval lengthening until a beat is dropped.
- Additional typical features of AV Wenckebach in Figure-4 include: i) The narrow QRS (Mobitz II usually has a wide QRS — because of its origin at a lower point in the conduction system); ii) Progressive shortening of the R-R interval within groups (the R-R intervals between beats #3-4; 6-7; and 9-10 — are shorter than the R-R intervals between beats #2-3; 5-6; 8-9); and, iii) The pause containing the dropped beat is less than twice the shortest R-R interval.
SUMMARY:
The rhythm in today's case is sinus (ie, regularly-occurring P waves) — with 2nd-degree AV block, Mobitz Type I ( = AV Wenckebach). Each of the 3-beat Wenckebach groups begins with a slightly accelerated junctional beat (which results in transient AV dissociation).
- The overall ventricular rate in today's tracing should be sufficient at ~60-80/minute for adequate perfusion.
- Although it is common for Mobitz I to be associated with acute inferior MI — the 12-lead ECG in today's case does not suggest any acute ST-T wave changes.
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LADDERGRAM:
To clarify the mechanism for today’s rhythm — I've derived a laddergram with step-by-step description of events. I begin with Figure-5.
- NOTE: For review of the Basics for HOW to read (and draw) laddergrams — See ECG Blog #188).
Figure-11: But the PR interval for the WHITE arrow P waves is clearly too short to conduct! (represented by WHITE butt ends drawn within the AV Nodal Tier). |
Final Advanced Point:
Did YOU notice in Figure-13 — that the QRS of each of the junctional beats ( = beats #2,5,8 and 11) is slightly taller than the QRS of the remaining 8 beats on this tracing?
- The above subtle observation can sometimes be a very helpful clue that these slightly different-looking beats are not sinus-conducted — but instead arise from the AV Node.
- The reason escape beats from the AV Node sometimes look slightly different than sinus-conducted beats — is that the precise site of origin within the AV Node from where these beats arise may be slightly different than the path within the AV Node through which sinus-conducted beats pass.
- To Emphasize: This clue is not needed for us to know that beats #2,5,8,11 in today's tracing are junctional beats — since it is obvious that the P waves preceding beats #2,5,8,11 are too close to the QRS to be conducted. But there are times when it will not be readily apparent if a given QRS complex represents a sinus-conducted beat vs junctional escape — in which case, the observation that QRS morphology differs slight may reveal which beats are junctional escape beats (See ECG Blog #63 — for a clinical example of this phenomenon!).
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Acknowledgment: My appreciation to ไกรสร เต็ง (from Bangkok, Thailand) for the case and this tracing.
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Related ECG Blog Posts to Today’s Case:
- ECG Blog #185 — Reviews my System for Rhythm Interpretation, using the Ps, Qs & 3R Approach.
- ECG Blog #205 — Reviews my System for 12-Lead ECG Interpretation.
- ECG Blog #245 — Reviews ECG diagnosis of LVH.
- ECG Blog #188 — Reviews how to read and draw Laddergrams (with LINKS to more than 90 laddergram cases — many with step-by-step sequential illustration).
- ECG Blog #192 — The 3 Causes of AV Dissociation.
- ECG Blog #191 — Reviews the difference between AV Dissociation vs Complete AV Block.
- ECG Blog #389 — ECG Blog #373 — and ECG Blog #344 — for review of some cases that illustrate "AV block problem-solving".
- ECG Blog #63 — Reviews a case of 2nd-Degree AV Block, Mobitz Type I with Junctional Escape Beats.
- ECG Blog #267 — Shows step-by-step Laddergrams, with derivation of a case of Mobitz I with more than a single possible explanation.
- ECG Blog #404 (ECG Video) — applies the Ps,Qs,3R Approach to a case of 2nd-degree AV Block ( = Mobitz I or Mobitz II or Neither).
- ECG Blog #405 (ECG Video) — applies the Ps,Qs,3R Approach to a case of AV Dissociation vs Complete AV Block (What's the difference?).
ADDENDUM (1/6/2024):
This 15-minute ECG Video (Media PEARL #52) — Reviews the 3 Types of 2nd-Degree AV Block — plus — the hard-to-define term of "high-grade" AV block. I supplement this material with the following 2 PDF handouts.
ECG Media PEARL #4 (4:30 minutes Audio): — takes a brief look at the AV Blocks — and focuses on WHEN to suspect Mobitz I. |
- Section 2F (6 pages = the "short" Answer) from my ECG-2014 Pocket Brain book provides quick written review of the AV Blocks.
- Section 20 (54 pages = the "long" Answer) from my ACLS-2013-Arrhythmias Expanded Version provides detailed discussion of WHAT the AV Blocks are — and what they are not!
ECG Media PEARL #75 (6:10 minutes Audio) — Reviews how to tell IF a Junctional (AV Nodal) Rhythm is pathologic or appropriate?
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